Abstract
In our research we exploit tomato and Arabidopsis as hosts for
the fungal pathogens Cladosporium fulvum and Verticillium
dahliae. Both species grow strictly extracellular without the
formation of feeding structures and are contained by so-called
receptor-like proteins, of which the type members are
Cf-resistance proteins, in incompatible interactions with
tomato. We have recently demonstrated that Ve1, but not Ve2
acts as a functional Verticillium resistance gene in tomato, and
started to unravel the genetics of Ve-mediated resistance
signaling. Downstream signaling components required by Ve1
for Verticillium resistance only partially overlap with those
required for Cf-mediated resistance against C. fulvum.
Furthermore, we have recently established the first model for
RLP-signaling in Arabidopsis, which allows studying the
genetics of RLP-mediated disease signaling in this host. Our
latest results will be discussed. Our analysis of host-pathogen
interactions with the extracellular pathogens C. fulvum and V.
dahliae also concerns the effectors that are secreted by these
pathogens while colonizing their hosts. Intriguingly, we
recently identified an effector from C. fulvum, designated
Ecp6, that is conserved between these two phylogenetically
unrelated species and which acts as a genuine virulence factor.
The role of Ecp6 during host colonization will be discussed
the fungal pathogens Cladosporium fulvum and Verticillium
dahliae. Both species grow strictly extracellular without the
formation of feeding structures and are contained by so-called
receptor-like proteins, of which the type members are
Cf-resistance proteins, in incompatible interactions with
tomato. We have recently demonstrated that Ve1, but not Ve2
acts as a functional Verticillium resistance gene in tomato, and
started to unravel the genetics of Ve-mediated resistance
signaling. Downstream signaling components required by Ve1
for Verticillium resistance only partially overlap with those
required for Cf-mediated resistance against C. fulvum.
Furthermore, we have recently established the first model for
RLP-signaling in Arabidopsis, which allows studying the
genetics of RLP-mediated disease signaling in this host. Our
latest results will be discussed. Our analysis of host-pathogen
interactions with the extracellular pathogens C. fulvum and V.
dahliae also concerns the effectors that are secreted by these
pathogens while colonizing their hosts. Intriguingly, we
recently identified an effector from C. fulvum, designated
Ecp6, that is conserved between these two phylogenetically
unrelated species and which acts as a genuine virulence factor.
The role of Ecp6 during host colonization will be discussed
| Original language | English |
|---|---|
| Title of host publication | ISMPMI International Congress abstracts, Quebec City, Canada, 19-23 July 2009 |
| Publisher | International Society for Molecular Plant-Microbe Interactions |
| Publication status | Published - 2009 |
| Event | XIV International Congress on Molecular Plant-Microbe Interactions, Quebec City, Canada - Duration: 19 Jul 2009 → 23 Jul 2009 |
Conference
| Conference | XIV International Congress on Molecular Plant-Microbe Interactions, Quebec City, Canada |
|---|---|
| Period | 19/07/09 → 23/07/09 |